IADR Abstract Archives

Denture Stomatitis Induces a Pro-inflammatory Cytokine Response

Denture stomatitis presents as erythema of the palatal mucosa and is an inflammatory condition associated with candidal colonisation of a complete or partial upper acrylic denture. Candida is recognised by pattern recognition receptors (PRRs) and leads to cytokine expression and a T-helper (Th) cell response. Whilst Th1 and Th17 responses are implicated with protection in animal models, their involvement in denture stomatitis has not been established.

Objectives: The aim of this study was to investigate Candida numbers and local cytokine expression in patients with or without denture stomatitis thereby indicating the different host immune response in these two groups.

Methods: Complete denture wearers with (n=42) and without (n=51) clinical signs of denture stomatitis were identified for inclusion in the study. Palatal inflammation was assessed using multiple clinical indices. Candida colonisation was assessed as CFU/cm2 from imprint cultures of the palate, fit surface of the denture and tongue. Levels of cytokines (IFNγ, IL-2, TNFα, IL-4, IL-5, IL-6, IL-10, IL-17a, IL-22 and TGF-beta1) were detected in palatal fluid samples. Cytokines were measured using a BDTM Cytometric Bead Array (CBA) human Th1/2/17 kit and single plex flex kits, and an Enzyme-linked immunosorbent assay (ELISA).

Results: Candida colonisation was significantly higher in the denture stomatitis group at all sites (palate, P=0.0301, denture, P=0.0113, tongue, P=0.0043) compared to the non-stomatitis group. Th1 (IFNγ, P=0.0004, TNFα, P=0.0009) and Th17 cytokines (IL-17a, P=0.0026, IL-22, P=0.0004) were also significantly increased in the stomatitis group. There was no significant increase in the Th2 cytokines (IL-4 and IL-5). A significant (P=0.0001) increase in TGF-beta1 was also evident which is a critical cytokine for Th17 cell differentiation.

Conclusions: In denture stomatitis subjects, increased Candida colonisation is associated with Th1 and Th17 responses by the host. This type of T cell response may protect the host from subsequent systemic infection.

Division: Pan European Region Meeting
Meeting: 2012 Pan European Region Meeting (Helisinki, Finland)
Location: Helisinki, Finland
Year: 2012
Final Presentation ID: 718
Abstract Category|Abstract Category(s): Scientific Groups
Authors
  • Rogers, Helen  ( School of Dentistry, Cardiff University, Cardiff, N/A, Wales )
  • Williams, David  ( School of Dentistry, Cardiff University, Cardiff, N/A, Wales )
  • Wei, Xiaoqing  ( School of Dentistry, Cardiff University, Cardiff, N/A, Wales )
  • Lewis, Michael  ( School of Dentistry, Cardiff University, Cardiff, N/A, Wales )
  • SESSION INFORMATION
    Poster Session
    Microbiology/Immunology II
    09/15/2012