IADR Abstract Archives

Expression Analysis of Inflammasome in Periodontal Tissues

Objectives:  In recent years, inflammatory responses are regarded as cellular and molecular bases of various biological reactions, such as homeostasis, tissue repair and regeneration. Not only exogenous pathogens but also endogenous stimuli are sensed by Toll-like receptors (TLRs) and inflammasome which is a cytoplasmic multiprotein complex consisting of caspase-1, apoptosis-associated speck-like protein containing a caspase recruitment domain (ASC), NOD-like receptors (NLRs), leading to the processing and secretion of the pro-inflammatory cytokines, IL-1β. Inflammasome cooperates with TLRs to mediate responses to pathogenic triggers and various stresses. In this study, we examined the expression of inflammasome in human periodontal-tissue component cells and during the course of differentiation of human periodontal ligament cells to hard-tissue forming cells in order to reveal the roles of inflammasome in periodontal tissues .

Methods: We analyzed the expression of the genes involved in inflammasome in human periodontal-tissue component cells (periodontal ligament cells, gingival epithelial cells, alveolar bone-derived osteoblasts and gingival fibroblasts) by RT-PCR. We immunohistochemically stained mouse maxilla to assess in vivoexpression of caspase-1 and ASC. Further, we cultured human periodontal ligament cells in mineralization inducing medium (αMEM supplemented with 10% FCS, 50 μg/ml Ascorbic acid and 10 mM β-glycerophosphate) for 30 days and real-time PCR was then performed to analyze the expression of the genes involved in inflammasome during the course of differentiation of human periodontal ligament cells.

Results: Human periodontal-tissue component cells showed expression of genes involved in inflammasome, such as caspase-1, ASC and NLR, pyrin domain-containing-3 (NLRP3). Caspase-1 and ASC expressions were also observed in mouse periodontal tissues. Expression of caspase-1, P2X7 and NLRP3 were increased during the course of differentiation of human periodontal ligament cells to hard-tissue forming cells,.

Conclusions: These results suggest that inflammasome may be involved in homeostasis and regeneration of periodontal tissues.

Division: Japanese Division Meeting
Meeting: 2012 Japanese Division Meeting (Niigata, Japan)
Location: Niigata, Japan
Year: 2012
Final Presentation ID:
Abstract Category|Abstract Category(s): Scientific Program
Authors
  • Yamaba, Satoko  ( Osaka Dental University, Suita, N/A, Japan )
  • Yamada, Satoru  ( Osaka Dental University, Suita, N/A, Japan )
  • Kajikawa, Tetsuhiro  ( Osaka Dental University, Suita, N/A, Japan )
  • Ozaki, Nobuhiro  ( Osaka Dental University, Suita, N/A, Japan )
  • Awata, Toshihito  ( Osaka Dental University, Suita, N/A, Japan )
  • Sakashita, Hiromi  ( Osaka Dental University, Suita, N/A, Japan )
  • Yanagita, Manabu  ( Osaka Dental University, Suita, N/A, Japan )
  • Kitamura, Masahiro  ( Osaka Dental University, Suita, N/A, Japan )
  • Murakami, Shinya  ( Osaka Dental University, Suita, N/A, Japan )
  • SESSION INFORMATION
    Periodontal Research