IADR Abstract Archives

Dental Pulp Stem Cells Promote Neural Regeneration Following Injury

Objective: Mesenchymal stem cells (MSC) are an emerging therapy option for damaged neural tissues. MSC may act indirectly to promote survival and regeneration of injured neurons through the release of neurotrophic factors. In this study we investigated the potential trophic benefits of dental pulp stem cells (DPSC) on injured retinal neurons.

Methods: DPSC and MSC were isolated from rat incisors and femurs, respectively. Stem cells at passage 2-4 were then tested in in vitro and in vivo adult rat retinal ganglion cell (RGC) injury models. Following co-culture with axotomized RGC, neuronal survival and neuritogenesis was analysed using βIII-tubulin immunostaining. For the in vivo studies, DPSC and MSC were transplanted into the vitreous body of the eye following surgically-induced optic nerve crush injury that axotomises RGC. Optical coherence tomography (OCT) was used to monitor RGC survival over time. RGC axonal regeneration was quantified 21 days post-lesion using immunohistochemical methods.

Results: When co-cultured with injured RGC, DPSC significantly enhanced cell survival and neurite outgrowth compared with RGC cultured alone or RGC co-cultured with bone marrow-derived MSC. The neuroprotective and pro-regenerative effects of the DPSC were abolished by Trk receptor blockers, suggesting involvement of paracrine neurotrophins produced by the DPSC. Consistent with these findings, enzyme-immunoassays indicated the secretion of significantly higher titres of the neurotrophins, nerve growth factor (NGF), brain-derived neurotrophic factor (BDNF) and NT-3 by DPSC than that produced by bone marrow MSC. Finally, in vivo intravitreal DPSC transplantation after optic nerve crush injury resulted in significant protection of RGC from apoptosis and regeneration of their axons, as analysed by Brn3a and growth-associated protein-43 immunostaining.

Conclusion: This study highlights the potential of DPSC as a cellular paracrine therapy for treating damaged neurons by providing a supportive neuroprotective/axogenic trophic environment.

Division: British Division Meeting
Meeting: 2013 British Division Meeting (Bath, England)
Location: Bath England
Year: 2013
Final Presentation ID: 22
Abstract Category|Abstract Category(s): Scientific Groups
Authors
  • Mead, Ben  ( University of Birmingham, Birmingham, N/A, England )
  • Berry, Martin  ( University of Birmingham, Birmingham, N/A, England )
  • Logan, Ann  ( University of Birmingham, Birmingham, N/A, England )
  • Leadbeater, Wendy  ( University of Birmingham, Birmingham, N/A, England )
  • Scheven, Ben A.  ( University of Birmingham, Birmingham, N/A, England )
  • SESSION INFORMATION
    Oral Session
    Tissue engineering
    09/09/2013