IADR Abstract Archives

Amelogenin-Mineral Interactions and the Control of Enamel Crystal Growth

Certain mutations in AMELX, the gene encoding the principle developing enamel matrix protein, amelogenin, result in enamel hypomineralisation in sex-linked Amelogenesis Imperfecta (AI), supporting suggestions that amelogenin has a role in directing mineral deposition and growth. Amelogenin is subject to extensive post-secretory proteolytic modification and mutations in the KLK4 gene (encoding the enamel serine protease kallikrein-4) are also known to cause autosomal recessive hypomaturation AI.

Objectives: The aim of the current study was to use atomic force microscopy (AFM) and dynamic force spectroscopy to characterise amelogenin-mineral crystal interactions at the molecular level and under in vivo like conditions.

Methods: Individual crystals were isolated from rat developing enamel using a selective extraction procedure to remove all native matrix protein. Crystals were imaged in the AFM in tapping mode under simulated enamel fluid at pH 7.4 in the presence of full-length amleogenin (rM179). rM179 was also covalently attached to chemically functionalised silicon-nitride AFM tips for dynamic force microscopy against individual crystal targets under similar conditions. Measurements of amelogenin-mineral interactions were calculated from the resulting force-distance curves.

Results: Amelogenin nanospheres were seen to align along crystal surfaces as described previously. Amelogenin-mineral associations were stable when exposed to increasing concentrations of phosphate buffer of up to 200 mM. The amelogenin-mineral dissociation constant (K0off) was found to be 0.08 s-1 at low loading rates when determined using dynamic force spectroscopy.

Conclusions: These results suggest a relatively stable parent amelogenin-mineral association. Post-secretory proteolytic processing of parent amelogenin is known to occur very rapidly in vivo and may be a necessary pre-requisite to permit normal crystal growth. Failure to remove protein may then result in hypomineralised enamel.

Funding: Supported by Wellcome programme grant no. 075945/Z/04/Z and BBSRC Grant no. 24/ABY08147


Division: British and Scandinavian Divisions Meeting
Meeting: 2007 British and Scandinavian Divisions Meeting (Durham, England)
Location: Durham, England
Year: 2007
Final Presentation ID: 33
Abstract Category|Abstract Category(s): Mineralized Tissue
Authors
  • Kirkham, Jennifer  ( University of Leeds, Leeds, N/A, United Kingdom )
  • Andreev, Igor  ( University of Leeds, Leeds, N/A, United Kingdom )
  • Brookes, Steven J.  ( University of Leeds, Leeds, N/A, United Kingdom )
  • Shore, Roger C.  ( University of Leeds, Leeds, N/A, United Kingdom )
  • Robinson, Colin  ( University of Leeds, Leeds, N/A, United Kingdom )
  • Smith, D. Alastair  ( University of Leeds, Leeds, N/A, United Kingdom )
  • SESSION INFORMATION
    Oral Session
    Mineralised Tissue: Mintig Prize ; Enamel
    04/03/2007