IADR Abstract Archives

The Role of BMP Signaling During Mandibular Skeletogenesis

Objectives: In order to develop novel strategies for regenerating skeletal tissues affected by disease and trauma, molecular mechanisms that drive the differentiation of mesenchymal stem cells into bone need to be identified. Previous avian embryonic studies demonstrate a time-dependent signal from mandibular epithelium that induces condensation and ossification of the underlying mesenchyme. Our goal was to ascertain the potential of TGFß family members to regulate this interaction, and thus the timing of skeletal differentiation. Specifically, we tested the ability of BMPs to augment and/or prematurely initiate bone formation.

Methods: Mandibles from quail embryos at Hamburger and Hamilton (HH) stage 23 were surgically extracted, placed on transwell membranes, and treated with agarose beads equilibrated in BMP4 or BMP7, using bovine serum albumin as contralateral controls. Mandibles were cultured in differentiation medium for three to six days and histologically assayed for bone. Representative sections were digitized and bone volume was estimated from the number of pixels comprising domains of stained matrix and condensations using the equation for a conical frustum: BV=1/3h[(Ai+Ai+1)+SQRT(AiAi+1)].

Results: Regardless of treatment, HH23 mandibles cultured for three and four days showed little to no histological evidence of bone. HH23 mandibles cultured for six days and treated with BMP4 or BMP7 exhibited an average 3-fold (p<.002) and 1.4-fold (p<.037) increase in bone condensation volume, respectively. Interestingly, four of those BMP4 mandibles developed no bone on the untreated side.

Conclusions: While time-dependent epithelial-mesenchymal interactions are known to regulate intramembranous ossification, our results reveal TGFß family members as candidate mediators of this process. The ability of exogenous BMPs to induce bone formation indicates that molecular-based therapies have strong potential to promote regeneration of skeletal tissues following trauma or degenerative skeletal disease.

Support: NIH/NIDCR/COHORT Training Grant T32/DE07306-09 to S.W.; NIDCR R03 DE014795-01 and R01 DE016402-01, and March of Dimes 5-FY04-26 to R.A.S.


Division: AADR/CADR Annual Meeting
Meeting: 2006 AADR/CADR Annual Meeting (Orlando, Florida)
Location: Orlando, Florida
Year: 2006
Final Presentation ID: 1225
Abstract Category|Abstract Category(s): Craniofacial Biology
Authors
  • Weston, Scott Jason  ( University of California - San Francisco, San Francisco, CA, USA )
  • Merrill, Amy Elizabeth  ( University of California - San Francisco, San Francisco, CA, USA )
  • Eames, Brian Frank  ( University of California - San Francisco, San Francisco, CA, USA )
  • Heath, Thayer  ( University of California - San Francisco, San Francisco, CA, USA )
  • Schneider, Richard A.  ( University of California - San Francisco, San Francisco, CA, USA )
  • SESSION INFORMATION
    Poster Session
    Development Biology of the Craniofacial Skeleton
    03/10/2006