IADR Abstract Archives

Oral Sarcomatoid Carcinoma - A Case Report and Molecular Study of Clonality

Objective: Sarcomatoid carcinoma is a dimorphic neoplasm with two components, squamous cell carcinoma and sarcomatoid spindle cells. It is an uncommon and aggressive malignancy, with a special predilection for the upper aerodigestive tract and oral cavity. Although immunohistochemical studies suggest that the sarcomatoid component represents a metaplastic alteration of the squamous carcinomatous component, there is a lack of genetic information to verify the clonality of the two morphological components. The purpose of this study is to investigate the clonality of the two components.

Method: Microsatellite analysis of loss of heterozygosity (LOH) has been used to identify clonality of tumors. This study analyzed one rare oral sarcomatoid squamous cell carcinoma by using both immunohistochemical straining (pankeratin and vimentin) and LOH assay with primers on chromosomes 3p14 (3p1234, 3p1300, 3p1228, 3p1285), 8p (8p261, 8p262, 8p264), 9p21 (9pINFA, 9p1751, 9p1748, 9p171), and 17p (tp53, 17p786, CHRNB).

Result: The patient died 10 weeks after the diagnosis. The carcinomatous component stained positive for keratin but negative for vimentin; whereas the sarcomatous component stained positive for vimentin with only occasional focal positivity for keratin. Both of the components showed an identical LOH pattern, loss at 3p14, 9p21 (regarded to be essential and early for development of oral squamous cell carcinoma) and 8p (associated with the aggressiveness of head and neck carcinoma). All of the markers for 17p were noninformative.

Conclusion: The study results for the first time provide genetic evidence supporting that the dimorphic components of the tumor are from the same molecular clone.


Division: AADR/CADR Annual Meeting
Meeting: 2003 AADR/CADR Annual Meeting (San Antonio, Texas)
Location: San Antonio, Texas
Year: 2003
Final Presentation ID: 398
Abstract Category|Abstract Category(s): Oral Medicine & Pathology
Authors
  • Poh, Catherine F.  ( The University of British Columbia, Vancouver, BC, Canada )
  • Li, Robert J.  ( Simon Fraser University, Burnaby, BC, Canada )
  • Priddy, Robert  ( The University of British Columbia, Vancouver, BC, Canada )
  • An, Ding  ( The University of British Columbia, Vancouver, BC, Canada )
  • Rosin, M.p.  ( British Columbia Cancer Agency, Vancouver, BC, Canada )
  • Zhang, Lewei  ( The University of British Columbia, Vancouver, BC, Canada )
  • SESSION INFORMATION
    Poster
    Carcinogenesis - Cell and Molecular Mechanisms
    03/13/2003