IADR Abstract Archives

Matrix Metalloproteinase Expression in Kaposi's Sarcoma

OBJECTIVE: Kaposi's sarcoma (KS), a common neoplasm in immunocompromised patients, is characterized by inflammation, angiogenesis, and proliferating spindle-shaped cells. Matrix metalloproteinases (MMPs) play a critical role in tumor invasion and metastasis, and in angiogenesis. Levels of MMPs expression, in KS lesions are unknown. To explore the role of MMPs in the invasive characteristics of KS, we examined the pattern of MMP, in vivo. METHODS:Histopathologic and immunolocalization studies were carried out using 5 Fm sections, of paraffin-embedded tumor tissue, and monoclonal antibodies specific for; MMP-1, -2, -3, -7, -9, -13, -14, and mast cell tryptase (Chemicon Intl., Temecula, CA), with Vector Nova Red substrate and Hematoxylin counterstain.

RESULTS: H & E stained sections revealed; prominent angiogenesis with proliferating vascular network associated with lymphocytes and plasma cells; and proliferating KS cells of mixed morphology with a predominant spindle cell component. Immunolocalization revealed the highest level of MMP expression was at sites of vascular invasion adjacent to vascular walls and in the presence of infiltrating inflammatory cells. Mast cell accumulation and degranulation plays a role at the tumor periphery and near vascular walls. High levels of MMPs expression was associated with morphological heterogeneity within the tumor cell population. KS cells with spindle morphology appeared to contribute with high levels of MMP-1, -2, -3, -9, and -13, and low level of MMP-7 and -14 expression. The stromal contribution, is very significant in the expression of MMP-1, -3, -9 and -14, but low MMP-2 and 13 at sites of inflammation and vascular invasion. Endothelial cells contribute by MMP-1, -3, -9, -14 and slight MMP-2, -7 and -13 expression. Conclusion: The in vivo expression of MMPs in KS is dictated by multifactorial interactions between KS cells, stromal fibroblasts and mast cells, inflammatory infiltrate, and finally by the angiogenic activity. (Supported in part by NIDCR grant DE-07258).


Division: AADR/CADR Annual Meeting
Meeting: 2003 AADR/CADR Annual Meeting (San Antonio, Texas)
Location: San Antonio, Texas
Year: 2003
Final Presentation ID: 1428
Abstract Category|Abstract Category(s): Periodontal Research - Pathogenesis
Authors
  • Harrison, Stephen M  ( University of Tennessee Health Science Center, Memphis, TN, USA )
  • Gaber, Lilian  ( , , , )
  • Haney, Lena  ( , , , )
  • Dabbous, Mustafa Kh  ( University of Tennessee, , N/A, )
  • SESSION INFORMATION
    Poster
    Periodontal disease/ Molecular Aspects of Fibroblasts
    03/14/2003