IADR Abstract Archives

Xylitol Stimulates the Secretory process via muscarinic receptor signaling pathway

Objective: Dry mouth characterized by reduced saliva secretion can be caused by various reasons such as medications, systemic or local diseases, or irradiation to the salivary glands. Xylitol, a well-known anti-caries agent, is known to enhance salivary secretion. However, the precise molecular mechanisms by which xylitol stimulate salivary secretion remains to be elucidated. Thus, we aimed to investigate the secretory effects of xylitol on human salivary gland (HSG) cell line, focusing on muscarinic type 3 receptor (M3R) signaling related pathways, which are critical in fluid secretion of salivary acinar cells.

Method: After HSG cells were treated with 3% of xylito, the expression of M3R and aquaporin 5(AQP5) was examined by PCR, western blotting and immunocytochemistry. To measure intracellular calcium release, cells were incubated with fluorescent Ca2+indicator Fluo-4 NW prior to agonist stimulation of cells in the presence or absence of xylitol and fluorescence intensity was detected at 485nm/535nm.

Result: Xylitol stimulated intracellular calcium release via M3R signaling pathway in HSG cells. The expression of M3R and AQP5 was also increased at mRNA and protein level by xylitol treatment. In addition, the induction of intracellular calcium release and the expression of M3R and AQP5 were inhibited by 4-DAMP, a selective M3R antagonist. Stimulation of muscarinic receptor induced by xylitol activated the internalization of M3R and subsequently trafficking of aquaporin5 to the plasma membrane.

Conclusion: Xylitol stimulated secretory process via muscarinic receptor signaling pathway and the trafficking of M3R and AQP5 in HSG cells.

Division: IADR/AADR/CADR General Session
Meeting: 2013 IADR/AADR/CADR General Session (Seattle, Washington)
Location: Seattle, Washington
Year: 2013
Final Presentation ID: 2241
Abstract Category|Abstract Category(s): Oral Medicine & Pathology
Authors
  • Park, Eunjoo  ( School of Dentistry, Pusan National University, Yangsan, N/A, South Korea )
  • Na, Heesam  ( Pusan National University, Yangsan-si, Gyeongsangnam-do, , South Korea )
  • Jeong, Sunghee  ( Pusan National University, Gyeongsangnam-do, , South Korea )
  • Lim, Hoisoon  ( Chonnam National University Hospital, Jeonnam, N/A, South Korea )
  • Cha, Seunghee  ( University of Florida, Gainesville, FL, USA )
  • Chung, Jin  ( Pusan National University, Yangsan-si, Gyeongsangnam-do, N/A, Republic Of Korea )
  • SESSION INFORMATION
    Poster Session
    Oral Mucositis and Therapy
    03/22/2013